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Part 4, 1726 nm lasers: Handling referred patients who are already on different treatment regimens
https://practicaldermatology.com/series/scientifically-speaking/1726-nm-lasers-five-dermatologists-discuss-the-lasers-benefits-and-drawbacks-for-treating-acne-and-other-skin-conditions/24342/Chapter 4 of 7: David Goldberg, MD, from New York, Emil Tanghetti, MD, from Sacramento, Jordan Wang, MD, from Philadelphia and New York and Naz Saedi, MD, from Philadelphia talk about handling referred patients who are already on different treatment regimens.What is Your Approach to Hair Loss in Women?
https://practicaldermatology.com/topics/aesthetics-cosmeceuticals/what-is-your-approach-to-hair-loss-in-women/19821/A seemingly simple question belies the complexity of managing hair loss. Dr. Friedman describes tactics from showing empathy to implementing treatments.Step Therapy Reform Advocates Seek to Spread Awareness of Proposed Law
https://practicaldermatology.com/issues/practicaldermatology-com-march-2025/step-therapy-reform-advocates-seek-to-spread-awareness-of-proposed-law/33160/The Safe Step Act would stop insurance companies under certain circumstances from requiring alternative medications before covering biologics.Active Vitiligo: What to Look For
https://practicaldermatology.com/series/updates-vitiligo/active-vitiligo-what-to-look-for/24156/John E. Harris, MD, founding director of the Autoimmune Therapeutics Institute at the University of Massachusetts Chan Medical School, talks with Practical Dermatology Chief Medical Editor Neal Bhatia, MD, about the clinical signs of active vitiligo.Advanced Laser Techniques from Cosmetic Surgery Forum 2019
https://practicaldermatology.com/topics/aesthetics-cosmeceuticals/advanced-laser-techniques-1/20125/What are the most promising energy-based options in laser technology. CSF faculty members, Jill Waibel, MD, Neil Sadick, MD, Manjula Jegasothy, MD, an Barry Lycka, MD offer fresh takes on the device field and share tips for optimizing outcomes.Let's Talk About Rosacea and Acne
https://practicaldermatology.com/conferences/scale-2023/acne-and-rosacea/20188/Hilary Baldwin, MD, shares pearls for treating papopustular rosacea, vascular rosacea, and acne.Antibiotics and Cosmetic Trends; Seysara Launches
https://practicaldermatology.com/series/dermwire-tv/dermwire-tv-antibiotics-and-cosmetics-trends-seysara-launches/19532/Dermatologists are prescribing fewer antibiotics to manage acne and rosacea. But, the use of antibiotics after surgical procedures is on the rise. More than one third of American adults are considering a cosmetic treatment in 2019, results of a RealSelf survey show. Seysara (sarecycline), a novel teClinical Conversations: Discover Dr J Scott Boswell’s Perspective on Redefining PN and AD Through a Neuroimmune Lens
https://practicaldermatology.com/programs/nemluvio-peer-network/clinical-conversations-discover-dr-j-scott-boswells-perspective-on-redefining-pn-and-ad-through-a-neuroimmune-lens/56421/In this Clinical Conversations newsletter, Dr Scott Boswell shares how reframing prurigo nodularis (PN) and atopic dermatitis (AD) as neuroimmune-driven conditions has shaped his clinical philosophy. He shares his 3‑step approach to patient communication, highlights real‑world treatment journeys inWhy Assessing Vitiligo Activity is Critical for Limiting Spread
https://practicaldermatology.com/conferences/maui-derm-2024/why-assessing-vitiligo-activity-is-critical-for-limiting-spread/20248/Chief Medical Editor Neal Bhatia, MD talks with “Mr. Vitiligo Himself,” John E. Harris, MD, PhD about the importance of determining whether vitiligo patients are active or stable in order to “shut off” spreading as soon as possible.How does chronic AD histopathology evolve from spongiosis-dominant lesions to lichenified, fibrotic plaques?
https://practicaldermatology.com/programs/dermatology-hub-neuroimmune-network/how-does-chronic-ad-histopathology-evolve-from-spongiosis-dominant-lesions-to-lichenified-fibrotic-plaques/48772/Christopher Bunick, MD, FAAD, discusses the roles of the six major cytokines that drive atopic dermatitis pathophysiology: IL-4, IL-13, IL-31, TSLP, IL-22, and IFN-γ.